eg https://mundoexchange.org/the-surprising-dangers-of-chopsticks/#:~:text=In%20our%20not%20too%20recent,harmful%20to%20your%20digestive%20system.
https://www.sciencedirect.com/science/article/pii/S0304389421031216#sec0010
Wood-based products leach at even higher rates than many plastics (that’s why Northwestern NUANCE labs don’t allow wood-based products in their cleanrooms)
this is like really relevant if u use paper bags to replace plastic containers…
you also can’t be sure that wood-based particles AREN’T being treated with plastics on the insides, like paper coffee cups
Pat25
#2
So paper coffee filters would be an issue also?
I’m getting more frustrated by this situation with each passing day.
It seems impossible to avoid (massive) exposure either way.
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AnUser
#3
The only reason I believe microplastics would be harmful is they are very small physical objects. So, it wouldn’t be any different from other ones including wood. You’d want physical objects that don’t cause very smaller physical objects. At the same time, isn’t many of the food we’re eating turned into very small physical objects, some of which we don’t use much and is just circulating?
Besides you have apoB particles of which atherosclerosis is the necessary cause. Without apoB you won’t have atheroschlerosis, which is the necessary step (and no microplastics stuck into plaque). But keeping them low enough seems to keep things in balance.
According to Claude and Perplexity.
10 mg / dl of apoB is 1.95 × 10^16 amount of apoB particles.
That’s 19,500,000,000,000,000 particles or 19.5 quadrillion circulating per dl of plasma now.
At 30 mg/dl you’d have 19,5 quadrillion x 3 per dl.
Atheroschlerosis, decrease healthspan, lifespan, really accelerate at higher quadrillion particle counts. We have insane amounts of evidence for this from MR, RCT’s, observational studies, mechanistic, etc, etc. The best we have for microplastics is them stuck in plaque caused by apoB, and plastic workers at probably crazy high doses!
Correct me if I’m wrong…
How many micro or nanoplastics are per dl of plasma?
I’m becoming more skeptical…
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Jjazz
#4
I would be infinitely more concerned with micoparticles from plastic than from wood. Our ancestors have always been in contact with natural microparticles, including those for wood, and our bodies and cells have evolved enzymes that are capable of degrading those substrates. Plastics are NOT something that we evolved with, and we thus do not have enzymes capable of depolymerizing them. So once a microplastic particle ends up in the lysosome of a cell, for example, it is probably never going to be degraded, leading to lysosomal dysfunction and downstream cellular dysfunction.
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